By A. M. Gotto Jr. (auth.), S. Lenzi, G. C. Descovich (eds.)
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Additional info for Atherosclerosis Clinical Evaluation and Therapy: Proceedings of the Fourth International Meeting on Atherosclerosis held in Bologna, Italy 23–25 November 1981
Another sub-group included 27 patients showing the clinical picture of ALe according to liver scan and needle-biopsy histology. Patients who had suffered oesophageal bleeding or abdominal collection of fluid were not included. A series of 70 males aged 30--65 was selected as 'controls' from a free-living population. 10 mg/dl, and BUN over 45 mg/ dl were excluded. Inclusion in the 'control' series was randomized according to the day of the first visit. Each patient's e, PL, P and TO were determined in total plasma and in the major lipoprotein classes (VLDL, IDL, LDL, HDL2 and HDL3)' Separation of the lipoprotein classes was performed using appropriate density gradients, according to the procedure of Havel et al.
These high levels were followed by a gradual decrease, and reached a minimum during summer (the months of July to September) and then increased slowly towards the winter. 5%. The variation in plasma triglyceride levels was distinctly different from that in plasma and lipoprotein cholesterol. In fact, it was almost an exact mirror image of that of plasma HDLC. The highest values were found during the summer and early autumn, the months of June-September, and the lowest during the winter. 4% of the highest.
Plasma lipids as collateral risk factors in coronary artery disease: A study of 371 males with chest pain. J. , 31, 337 Stout, R. W. (1981). Blood glucose and atherosclerosis. Arteriosclerosis, 1, 227 Verdery, R. B. and Nichols, A. V. (1975). Arrangement of lipids and proteins in human serum high density lipoproteins: A proposed model. Chem. Phys. Lipids, 14, 123 Weisgraber, K. , Rail, S. , Jr. and Mahley, R. W. (1981). HumanE apoprotein heterogeneity: Cysteine-arginine interchanges in the amino acid sequence of the apoE isoforms.